学会発表(国際)
Matsuzaki G., Okita Y., Umemura M.: Innate protective immunity of non-hematopoietic cells against Listeria monocytogenes infection induced by IL-17 and IL-22. (The 12th Japan-Korea International Symposium on Microbiology (XII-JKISM), Tower Hall Funabori, Tokyo Japan, 2014/03/24-25)
Matsuzaki G., Okita Y., Shiono T., Hamada S., Umemura M.: Mechanism of interleukin (IL)-17A-mediated enhancement of protective immunity against Listeria monocytogenes infection. (15th International Congress of Immunology, Milan Italy, 2013/08/22-27)
Fukui M., Umemura M., Matsuzaki G.: Eicosapentaenoic acid induces apoptosis in human pancreatic cancer cells. Role of ROS accumulation, caspase 8 activation, and autophagy induction. (IMMUNOLOGY 2013, Hawaii Convention Center, Honolulu, Hawaii, USA, 2013/05/03-07)
Umemura M., Touyama S., Fukui M., Yahagi A., Nakae S., Iwakura Y., Matsuzaki G.: Involvement of IL-17A-producing TCR γδ T cells in late protective immunity against pulmonary mycobacterial infection. (IMMUNOLOGY 2013, Hawaii Convention Center, Honolulu, Hawaii, USA, 2013/05/03-07)
Matsuzaki G., Okamoto-Yoshida Y., Umemura M.: Role of IL-17-producing gamma/delta T cells in granuloma formation of mycobacteria-infected lung. (International Union of Microbiological Societies 2011 Congress, Sapporo Convention Center, Sapporo, Japan, 2011/09/06-10)
Umemura M., Okamoto Y., Yahagi A., Toma N, Nakae S., Iwakura Y., Matsuzaki G.: Essential role of interleukin-17A in the formation of a mycobacterial infection-induced granuloma in the lung. (International Union of Microbiological Societies 2011 Congress, Sapporo Convention Center, Sapporo, Japan, 2011/09/06-10)
Umemura M., Okamoto Y., Yahagi A., Toma N., Nakae S., Iwakura Y., Matsuzaki G.: The role of IL-17A producing TCRγδT cells in the granuloma formation in the lung during mycobacterial infection. (76th Japanese Society of Interferon & Cytokine research and the 19th International Symposium of Molecular Cell Biology of Macrophages, ANA Gate Tower Hotel Osaka, Osaka, Japan, 2011/05/25-27)
Arakawa T, Miyata T, Harakuni T. A new vaccine platform technology for improved protection against malaria parasite infection. International Meeting of the Federation of Korean Microbiological Societies 14-15 Oct., 2010, Seoul, Korea
Arakawa T, Miyata T. Harakuni, T. A new vaccine platform technology for improved protection against malaria parasite infection. The control of emerging infectious diseases. 13 Oct., 2010, Daejeong, Korea.
Yahagi A., Umemura M., Tamura T., Kariyone A., Begum D., Yoshida-Okamoto Y., Ishihara K., Takatsu K., Matsuzaki G.: Suppressed induction of mycobacterial antigen-specific Th1-type CD4+ T cells in the lung after pulmonary mycobacterial infection. (14th Inteernational Congress of Immunology, Kobe, Japan, 23-27 Aug. 2010.)
Umemura M., Yoshida-Okamoto Y., Yahagi A., Nakane S., Iwakura Y., Matsuzaki G.: Essential role of interleukin-17A in the formation of mycobacterial infection-induced granuloma in the lung. (14th Inteernational Congress of Immunology, Kobe, Japan, 23-27 Aug. 2010.)
Miyata T. Ikehara A, Harakuni T, Tsuboi T, Sattabongkot J, Tachibana M, Torii M, Matsuzaki G. Arakawa T. “Tricomponent Immunopotentiating System”, a novel vaccine platform technology. 14th Inteernational Congress of Immunology, Kobe, Japan, 23-27 Aug. 2010.
Umemura M., Okamoto Y., Yahagi A., Nakae S., Iwakura Y., Matsuzaki G.: Essential role of interleukin-17A in the formation of a mycobacterial infection-induced granuloma in the lung. (4th γδT cell Conference, Kiel, Germany, 19-21 May 2010.)
Umemura M., Yahagi A., Okamoto Y., Begum D.M., Matsuzaki G.: Role of IL-17 producing T cells in the lung of pulmonary mycobacterial infection. (Keystone Symposia, Keystone Colorado USA, 2008/02)
Umemura M., Yahagi A., Okamoto Y., Nakae S., Iwakura Y., Matsuzaki G.: IL-17-mediated regulation of innate and acquired immune response against pulmonary mycobacterial infection. (13th International Congress of Mucosal Immunology, Tokyo, Japan, 2007/07)
Umemura M., Yahagi A., Hamada S., Matsuzaki G.: IL-17-mediated regulation of innate and acquired immune response against pulmonary mycobacterial infection. (Annual Meeting of The American Association of Immunologists, Boston MA USA, 2006/05)